Fig. 1. The underlying mechanism of anoxic depolarization and glutamate excitotoxicity after acute cerebral ischemia. AMPA, glutamatergic α-amino-3-hydroxy-5-methyl-4-isoxazole propionic acid receptors; Ca2+, calcium ion; Cl-, chloride ion; Glu, glutamate; Na+, sodium ion; NMDA, N-methyl-D-aspartate receptor; VDCC, voltage-dependent calcium channels.
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